Hans Huitink
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Hans Huitink
@airwaymxacademy.bsky.social
Anesthesiologist🇳🇱Founder Airway Management Academy non-profit airway teaching #AAF25 | Mobile Critical Care Support😷🚑🩺 | AirAmbulance🛩 @AirwayTriageApp
Reposted by Hans Huitink
On balance, the marginal utility of cricoid pressure (in this position) is outweighed by its potential disadvantages. Seldinger’s cadaveric studies (in steep head down) have utility in demonstrating pressure is transmitted to the oesophagus, but..
November 15, 2025 at 9:02 PM
Reposted by Hans Huitink
Having said that in that first case of hyperacute onset, neurological symptoms started almost immediately post op and peaked after two days. That would be quite the coincidence.
October 21, 2025 at 8:35 PM
Maybe high dose corticosteroids?
October 23, 2025 at 6:27 PM
Reposted by Hans Huitink
A more plausible mechanism would be the inflammatory effects of surgery, rather than toxicity/triggering from a spinal. I feel like the spinal is an innocent bystander.

In this SR of case reports, the majority of surgeries are spine and cardiac.

pmc.ncbi.nlm.nih.gov/articles/PMC...
Guillain-Barré syndrome after surgery: a literature review
Guillain-Barré syndrome (GBS) is a rare postoperative complication that is sometimes characterized by serious motor weakness and prolonged weaning from mechanical ventilation. Although the exact natur...
pmc.ncbi.nlm.nih.gov
October 21, 2025 at 10:59 PM
Fully agree
October 23, 2025 at 6:26 PM
Reposted by Hans Huitink
I think it is a coincidence. GBS can occur within 4 weeks of campylobacter infection. Preop diarrhoea can be common in some populations (eg bariatrics ~ nearly 10%), and it isn’t usually asked about.
October 21, 2025 at 10:59 PM
Reposted by Hans Huitink
Have any authors proposed a mechanism whereby a central drug exposure creates a peripheral neuropathy?
October 21, 2025 at 9:31 PM
“Do not intubate in the “coffin position””. Quote Dr Richard Levitan
October 21, 2025 at 9:22 AM