Comprehensive Echocardiographic Assessment of Right Ventricular Afterload and Right Ventricular–Pulmonary Arterial Coupling in the Intensive Care Unit
CCR Journal Watch
https://criticalcarereviews.com/latest-evidence/journal-watch
CCR Journal Watch
https://criticalcarereviews.com/latest-evidence/journal-watch
October 31, 2026 at 8:36 PM
Comprehensive Echocardiographic Assessment of Right Ventricular Afterload and Right Ventricular–Pulmonary Arterial Coupling in the Intensive Care Unit
CCR Journal Watch
https://criticalcarereviews.com/latest-evidence/journal-watch
CCR Journal Watch
https://criticalcarereviews.com/latest-evidence/journal-watch
Pharmacology of vasopressors & inotropes during #ECMO
🫀hemodynamic dysregulation
🩸unique considerations during ECLS: in-parallel circulation + afterload mismatch, concomitant circulatory devices + unloading
💉 catecholamines & derivatives
💉 non-catecholamines
#FOAMcc #FOAMecmo
🔓 rdcu.be/MZ5Q1syp79Ua
🫀hemodynamic dysregulation
🩸unique considerations during ECLS: in-parallel circulation + afterload mismatch, concomitant circulatory devices + unloading
💉 catecholamines & derivatives
💉 non-catecholamines
#FOAMcc #FOAMecmo
🔓 rdcu.be/MZ5Q1syp79Ua
October 23, 2026 at 9:25 AM
Pharmacology of vasopressors & inotropes during #ECMO
🫀hemodynamic dysregulation
🩸unique considerations during ECLS: in-parallel circulation + afterload mismatch, concomitant circulatory devices + unloading
💉 catecholamines & derivatives
💉 non-catecholamines
#FOAMcc #FOAMecmo
🔓 rdcu.be/MZ5Q1syp79Ua
🫀hemodynamic dysregulation
🩸unique considerations during ECLS: in-parallel circulation + afterload mismatch, concomitant circulatory devices + unloading
💉 catecholamines & derivatives
💉 non-catecholamines
#FOAMcc #FOAMecmo
🔓 rdcu.be/MZ5Q1syp79Ua
I think measures of RV afterload would still be instructive but I agree that I don’t except VV to really offload, either
October 22, 2026 at 12:47 PM
I think measures of RV afterload would still be instructive but I agree that I don’t except VV to really offload, either
The only times I recommend dobutamine in selsis, is when I can’t get the treating physician tontop the vasopressin which is too much afterload with too little inotropy to match.
October 16, 2026 at 6:26 PM
The only times I recommend dobutamine in selsis, is when I can’t get the treating physician tontop the vasopressin which is too much afterload with too little inotropy to match.
I would not think that VV ECMO would really hide mechanical RV afterload. Yes, your afterload due to physiologic factors like O2 and CO2 should be minimal but the blood still has to pass through the RV.
October 5, 2026 at 1:36 PM
I would not think that VV ECMO would really hide mechanical RV afterload. Yes, your afterload due to physiologic factors like O2 and CO2 should be minimal but the blood still has to pass through the RV.
8️⃣ Is the flow lower than expected?
Walk through the sequence:
Position → Preload → Afterload → Mechanical hardware
Don't jump straight to device escalation when the afterload is sky-high or the inlet is displaced.
Walk through the sequence:
Position → Preload → Afterload → Mechanical hardware
Don't jump straight to device escalation when the afterload is sky-high or the inlet is displaced.
October 3, 2026 at 8:19 PM
8️⃣ Is the flow lower than expected?
Walk through the sequence:
Position → Preload → Afterload → Mechanical hardware
Don't jump straight to device escalation when the afterload is sky-high or the inlet is displaced.
Walk through the sequence:
Position → Preload → Afterload → Mechanical hardware
Don't jump straight to device escalation when the afterload is sky-high or the inlet is displaced.
📝Editorial perspective: Is less sedation always better in the CICU?
Deep sedation may offer physiologic benefits in selected cardiac patients, including afterload reduction & arrhythmia control. Maybe the question is: Who needs deeper sedation, and when?
🔗 bit.ly/4dHsKJs
Deep sedation may offer physiologic benefits in selected cardiac patients, including afterload reduction & arrhythmia control. Maybe the question is: Who needs deeper sedation, and when?
🔗 bit.ly/4dHsKJs
September 22, 2026 at 6:37 PM
📝Editorial perspective: Is less sedation always better in the CICU?
Deep sedation may offer physiologic benefits in selected cardiac patients, including afterload reduction & arrhythmia control. Maybe the question is: Who needs deeper sedation, and when?
🔗 bit.ly/4dHsKJs
Deep sedation may offer physiologic benefits in selected cardiac patients, including afterload reduction & arrhythmia control. Maybe the question is: Who needs deeper sedation, and when?
🔗 bit.ly/4dHsKJs
Great points, I really see IABP as a mechanical afterload reductor (great effect in dilated LVs and especially in dynamic mitral regurgitations), the benefit in increased DAP is more theoretical than real I think
September 10, 2026 at 7:42 PM
Great points, I really see IABP as a mechanical afterload reductor (great effect in dilated LVs and especially in dynamic mitral regurgitations), the benefit in increased DAP is more theoretical than real I think
This case emphasizes the interplay between psychological factors and the MV, demonstrating how emotional stress and anxiety can lead to quick alteration in MV morphology and function due to increased sympathetic tone and abrupt changes in afterload.
February 18, 2025 at 10:21 PM
This case emphasizes the interplay between psychological factors and the MV, demonstrating how emotional stress and anxiety can lead to quick alteration in MV morphology and function due to increased sympathetic tone and abrupt changes in afterload.
Ineffective right ventricular adaptation to increasing pulmonary arterial afterload in pulmonary vascular disease significantly contributes to morbidity and mortality.
We examine whether echocardiographic RV-PA coupling metrics correlate with invasive pressure-volume loops. bit.ly/4aQXt4d
We examine whether echocardiographic RV-PA coupling metrics correlate with invasive pressure-volume loops. bit.ly/4aQXt4d
February 6, 2025 at 7:52 PM
Ineffective right ventricular adaptation to increasing pulmonary arterial afterload in pulmonary vascular disease significantly contributes to morbidity and mortality.
We examine whether echocardiographic RV-PA coupling metrics correlate with invasive pressure-volume loops. bit.ly/4aQXt4d
We examine whether echocardiographic RV-PA coupling metrics correlate with invasive pressure-volume loops. bit.ly/4aQXt4d
🔟 Frank-Starling Mechanism 📈
The heart adapts to volume changes!
⬆️ Preload = ⬆️ Stretch = Stronger contraction = ⬆️ Stroke volume.
Too much stretch? 💔 Heart failure risk increases!
The heart adapts to volume changes!
⬆️ Preload = ⬆️ Stretch = Stronger contraction = ⬆️ Stroke volume.
Too much stretch? 💔 Heart failure risk increases!
February 25, 2025 at 7:37 AM
🔟 Frank-Starling Mechanism 📈
The heart adapts to volume changes!
⬆️ Preload = ⬆️ Stretch = Stronger contraction = ⬆️ Stroke volume.
Too much stretch? 💔 Heart failure risk increases!
The heart adapts to volume changes!
⬆️ Preload = ⬆️ Stretch = Stronger contraction = ⬆️ Stroke volume.
Too much stretch? 💔 Heart failure risk increases!
9️⃣ Haemodynamics: The Heart in Action 🏃♂️💨
🔹 Preload 📏 = Stretch of ventricles before contraction
🔹 Afterload 🏋️♂️ = Resistance the LV must push against
🔹 Contractility 💥 = Strength of the heart’s contraction
🔹 Preload 📏 = Stretch of ventricles before contraction
🔹 Afterload 🏋️♂️ = Resistance the LV must push against
🔹 Contractility 💥 = Strength of the heart’s contraction
February 25, 2025 at 7:37 AM
9️⃣ Haemodynamics: The Heart in Action 🏃♂️💨
🔹 Preload 📏 = Stretch of ventricles before contraction
🔹 Afterload 🏋️♂️ = Resistance the LV must push against
🔹 Contractility 💥 = Strength of the heart’s contraction
🔹 Preload 📏 = Stretch of ventricles before contraction
🔹 Afterload 🏋️♂️ = Resistance the LV must push against
🔹 Contractility 💥 = Strength of the heart’s contraction
NEET PG cardiovascular physiology cardiac output variables and pressure volume loops checklist.#NEETPG2026 #INICET #NEXTExam #HighYieldReview #Physiology #CardiovascularPhysiology #CardiacOutput #StrokeVolume #Preload #Afterload #Contractility #FrankStarling #FickPrinciple #PVLoops #Hemodynamics
Cardiac output variables
Cardiac output parameters form the core of cardiovascular physiology on the NEET PG exam floor. Transition beyond basic definitions and master the precise mechanics—from compliance changes in myocardial loops to complex clinical calculations—that govern multi-system pharmacology and critical care vignettes.
mymedschool.org
June 24, 2026 at 12:20 PM
NEET PG cardiovascular physiology cardiac output variables and pressure volume loops checklist.#NEETPG2026 #INICET #NEXTExam #HighYieldReview #Physiology #CardiovascularPhysiology #CardiacOutput #StrokeVolume #Preload #Afterload #Contractility #FrankStarling #FickPrinciple #PVLoops #Hemodynamics
This H5P drag-and-drop activity reinforces a core cardiovascular physiology concept: the determinants of stroke volume. By matching contractility, preload, and afterload with their effects, learners strengthen conceptual understanding essential for Step 1, clinical reasoning, and cardiac output anal
U01.07.006 Cardiac output variables
Drag the words into the correct boxes, matching the effect on stroke volume. Stroke volume is affected by contractility, preload, and afterload. Learners must identify how each factor increases or decreases stroke volume.
mymedschool.org
February 16, 2026 at 1:03 AM
This H5P drag-and-drop activity reinforces a core cardiovascular physiology concept: the determinants of stroke volume. By matching contractility, preload, and afterload with their effects, learners strengthen conceptual understanding essential for Step 1, clinical reasoning, and cardiac output anal
Cardiac output, the volume of blood pumped by the heart per minute, is determined by stroke volume and heart rate. Stroke volume itself depends on preload, afterload, and myocardial contractility, making these variables central to cardiovascular physiology. In this multiple-choice question, you will
U01.07.007 Cardiac output variables
MCQ on cardiac output variables—test your knowledge of how heart rate, stroke volume, preload, afterload, and contractility interact to regulate cardiac performance
mymedschool.org
September 17, 2025 at 3:47 PM
Cardiac output, the volume of blood pumped by the heart per minute, is determined by stroke volume and heart rate. Stroke volume itself depends on preload, afterload, and myocardial contractility, making these variables central to cardiovascular physiology. In this multiple-choice question, you will
Cardiac output is shaped not only by physiological variables like preload, afterload, and contractility but also by pharmacological interventions used in cardiovascular medicine. Drugs such as nitrates reduce preload through venodilation, ACE inhibitors and arterial vasodilators lower afterload, diu
U01.07.007 Cardiac output variables
Interactive drag-and-drop activity on cardiac output variables and pharmacology—identify how drugs such as diuretics, nitrates, ACE inhibitors, beta-blockers, and vasodilators alter preload, afterload, and overall cardiac output.
mymedschool.org
September 17, 2025 at 3:08 PM
Cardiac output is shaped not only by physiological variables like preload, afterload, and contractility but also by pharmacological interventions used in cardiovascular medicine. Drugs such as nitrates reduce preload through venodilation, ACE inhibitors and arterial vasodilators lower afterload, diu
Cardiac output, the volume of blood the heart pumps per minute, is determined primarily by stroke volume and heart rate, both of which are influenced by physiological factors such as preload, afterload, and myocardial contractility. In this drag-and-drop activity, you will identify each variable, un
U01.07.007 Cardiac output variables
Interactive drag-and-drop question on cardiac output variables—identify stroke volume, heart rate, preload, afterload, and contractility, and understand how changes in these factors influence cardiac output and overall cardiovascular function.
mymedschool.org
September 16, 2025 at 5:26 PM
Cardiac output, the volume of blood the heart pumps per minute, is determined primarily by stroke volume and heart rate, both of which are influenced by physiological factors such as preload, afterload, and myocardial contractility. In this drag-and-drop activity, you will identify each variable, un
Cardiac output (CO) is the product of heart rate (HR) and stroke volume (SV). Stroke volume is influenced by preload (ventricular filling), afterload (resistance to ejection), and contractility (myocardial strength). In this activity, drag each variable to its effect on CO, recognize how physiologic
U01.07.007 Cardiac output variables
Interactive drag-and-drop activity to understand cardiac output variables—identify factors affecting stroke volume, heart rate, and their clinical implications in cardiac physiology.
mymedschool.org
September 16, 2025 at 4:34 PM
Cardiac output (CO) is the product of heart rate (HR) and stroke volume (SV). Stroke volume is influenced by preload (ventricular filling), afterload (resistance to ejection), and contractility (myocardial strength). In this activity, drag each variable to its effect on CO, recognize how physiologic
• IMPELLA PHYSIOLOGY
“Small loop, big impact”
Graph depicting LV pressure-volume loop under Impella support, reduced end-diastolic volume and pressure, decreased afterload, and a leftward shift of the loop, indicating LV unloading.
👉Learn more: IMPELLA PHYSIOLOGY youtu.be/JGXevZWm61E
#impella
“Small loop, big impact”
Graph depicting LV pressure-volume loop under Impella support, reduced end-diastolic volume and pressure, decreased afterload, and a leftward shift of the loop, indicating LV unloading.
👉Learn more: IMPELLA PHYSIOLOGY youtu.be/JGXevZWm61E
#impella
August 11, 2025 at 7:34 AM
• IMPELLA PHYSIOLOGY
“Small loop, big impact”
Graph depicting LV pressure-volume loop under Impella support, reduced end-diastolic volume and pressure, decreased afterload, and a leftward shift of the loop, indicating LV unloading.
👉Learn more: IMPELLA PHYSIOLOGY youtu.be/JGXevZWm61E
#impella
“Small loop, big impact”
Graph depicting LV pressure-volume loop under Impella support, reduced end-diastolic volume and pressure, decreased afterload, and a leftward shift of the loop, indicating LV unloading.
👉Learn more: IMPELLA PHYSIOLOGY youtu.be/JGXevZWm61E
#impella
Understanding Aortic Stenosis 🔄
Aortic stenosis increases afterload, leading to LV hypertrophy and, over time, heart failure. Watch for syncope, angina, and dyspnea (SAD)—classic symptoms of severe AS. TAVR or SAVR is the definitive fix! #TAVR #cardiosky
Aortic stenosis increases afterload, leading to LV hypertrophy and, over time, heart failure. Watch for syncope, angina, and dyspnea (SAD)—classic symptoms of severe AS. TAVR or SAVR is the definitive fix! #TAVR #cardiosky
February 25, 2025 at 4:47 PM
Understanding Aortic Stenosis 🔄
Aortic stenosis increases afterload, leading to LV hypertrophy and, over time, heart failure. Watch for syncope, angina, and dyspnea (SAD)—classic symptoms of severe AS. TAVR or SAVR is the definitive fix! #TAVR #cardiosky
Aortic stenosis increases afterload, leading to LV hypertrophy and, over time, heart failure. Watch for syncope, angina, and dyspnea (SAD)—classic symptoms of severe AS. TAVR or SAVR is the definitive fix! #TAVR #cardiosky
"Cardiac System Overview - Heart Chambers, Valves, Layers and Cardiac Output" ( www.vhtc.org/2026/06/card... )
Cardiac System Overview - Heart Chambers, Valves, Layers and Cardiac Output
Cardiac System Overview - Heart chambers, valves, heart layers, pericardium, blood flow, cardiac output, preload, afterload, and ejection fraction.
www.vhtc.org
June 22, 2026 at 11:21 AM
"Cardiac System Overview - Heart Chambers, Valves, Layers and Cardiac Output" ( www.vhtc.org/2026/06/card... )
This can also back up the pressure to your heart, which is called Afterload. It's the blood pressure your heart has to push against each time it pumps.
If it can't adequately pump against that pressure, blood effectively starts to pool in the chambers of your heart.
If it can't adequately pump against that pressure, blood effectively starts to pool in the chambers of your heart.
July 16, 2025 at 3:27 PM
This can also back up the pressure to your heart, which is called Afterload. It's the blood pressure your heart has to push against each time it pumps.
If it can't adequately pump against that pressure, blood effectively starts to pool in the chambers of your heart.
If it can't adequately pump against that pressure, blood effectively starts to pool in the chambers of your heart.
Fingers crossed.
Those aren't the best of numbers but in comparison to before, they're a great improvement.
In case you were interested in the 145 number, that's called the afterload, which is the pressure in the arteries that the heart has to push against to push blood out of it.
Those aren't the best of numbers but in comparison to before, they're a great improvement.
In case you were interested in the 145 number, that's called the afterload, which is the pressure in the arteries that the heart has to push against to push blood out of it.
July 4, 2025 at 11:43 PM
Fingers crossed.
Those aren't the best of numbers but in comparison to before, they're a great improvement.
In case you were interested in the 145 number, that's called the afterload, which is the pressure in the arteries that the heart has to push against to push blood out of it.
Those aren't the best of numbers but in comparison to before, they're a great improvement.
In case you were interested in the 145 number, that's called the afterload, which is the pressure in the arteries that the heart has to push against to push blood out of it.
Our review on sexual dimorphisms in RV function is out! My co-authors are a part of two amazing collaborations that have made this year the most fun year of research yet in my career. #teamscience @timlahm.bsky.social @atscommunity.bsky.social @ats-pc.bsky.social
Sex-dependent pathophysiology and therapeutic considerations in right heart disease
Right ventricular (RV) adaptation to the increased afterload in the setting of pulmonary hypertension (PH) and other cardiac and pulmonary vascular co…
www.sciencedirect.com
March 7, 2025 at 7:52 PM
Our review on sexual dimorphisms in RV function is out! My co-authors are a part of two amazing collaborations that have made this year the most fun year of research yet in my career. #teamscience @timlahm.bsky.social @atscommunity.bsky.social @ats-pc.bsky.social