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wikipathways.fosstodon.org.ap.brid.gy
April 2026 WikiPathways update: 71 edits by 7 contributorsin and 2 new pathways the last month. More news at https://github.com/wikipathways/wikipathways-help/discussions/231 Accessible via #webservices, #rstats, #PathVisio and #Cytoscape. https://www.wikipathways.org/index.php/Download_Pathways […]
Original post on fosstodon.org
fosstodon.org
April 12, 2026 at 6:27 AM
wikipathways.fosstodon.org.ap.brid.gy
March 2026 WikiPathways update: 45 edits by 5 contributors in the last month. More news at https://github.com/wikipathways/wikipathways-help/discussions/227

Accessible via #webservices, #rstats, #PathVisio and #Cytoscape. Supported by #AWSOpen […]
Original post on fosstodon.org
fosstodon.org
March 10, 2026 at 9:31 PM
wikipathways.fosstodon.org.ap.brid.gy
February 2026 WikiPathways update: 95 edits by 9 contributorsin the last month. More news at https://github.com/wikipathways/wikipathways-help/discussions/221 Accessible via #webservices, #rstats, #PathVisio and #Cytoscape. Supported by #AWSOpen […]
Original post on fosstodon.org
fosstodon.org
February 12, 2026 at 5:59 AM
wikipathways.fosstodon.org.ap.brid.gy
January 2026 WikiPathways update: 134 edits by 3 contributorsin the last month. More news at https://github.com/wikipathways/wikipathways-help/discussions/213 Accessible via #webservices, #rstats, #PathVisio and #Cytoscape. Supported by #AWSOpen […]
Original post on fosstodon.org
fosstodon.org
January 11, 2026 at 12:14 PM
biohackrxiv.fediscience.org.ap.brid.gy
"QPX: Pathway analysis environment" https://doi.org/10.37044/osf.io/m37f2_v1

"Building on our work at DBCLS BioHackathon 2023 (BH23), where we introduced QPX andpromoted pathway modeling with WikiPathways (Pico et al., 2008) using PathVisio (Kutmon etal […]

[Original post on fediscience.org]
Screenshot of the linked BioHackrXiv preprint, showing the top half of a preprint PDF page, showing the BioHackrXiv logo from the template, a table at the top listing a Arabidopsis thaliana pathway, and below that part of Figure 1 showing a "[p]athway diagram for for Caffeine synthesis inCoffea arabica. This diagram is already published in WikiPathways at https://www.wikipathways.org/pathways/WP5586.html".
January 6, 2026 at 9:01 PM
bridgedb.fosstodon.org.ap.brid.gy
a new BridgeDb metabolite ID mapping database has been released, compatible with PathVisio 3.3: https://doi.org/10.6084/m9.figshare.30993322
January 3, 2026 at 8:28 AM
wikipathways.fosstodon.org.ap.brid.gy
December 2025 WikiPathways release: 840 edits by 8 contributors and 9 new pathways. Accessible via #webservices, #rstats, #PathVisio and #Cytoscape.

https://www.wikipathways.org/#download

#bioinformatics #elixirnl
WikiPathways WP5609: Key metabolic pathways in melanoma, glycolytic pathway, TCA cycle, glutamine metabolism, and oxidative phosphorylation, with potential therapeutic targets and inhibitors. Inhibitors targeting critical metabolic nodes are outlined in teal. This pathway is based on Figure 1 in Shen et al. WikiPathways WP5606: Cis- and trans-acting regulators shape gene expression within the β-globin cluster. The LDB1 complex (LDB1/LMO2/GATA1/TAL1) binds both the locus control region (LCR) and globin promoters, promoting chromatin looping to activate these genes—interactions that may be influenced by cis-acting variants linked to HbS haplotypes. Across the HBB gene cluster and its surrounding regions, BCL11A and ZBTB7A (LRF) binding sites are present, represented by red and blue stars. Each of these transcription factors recruits its own NuRD complex. MYB regulates HbF expression directly and also indirectly through KLF1 and BCL11A. Repression of the HbF genes is indicated by dashed lines. Inspired by figure 1 in Habara et al. (2017). WikiPathways WP5604: This pathway shows the molecular pathophysiology of sickle cell disease. (A) A single–nucleotide variant in the β-globin gene replaces glutamic acid with valine at position 6 of the β-globin chain. Upon deoxygenation, the resulting hemoglobin S (HbS) molecules polymerize into rigid fibers, driving erythrocyte sickling (clockwise). (B) Sickled cells cause impaired blood rheology and enhanced adhesion of erythrocytes to neutrophils, platelets, and the endothelium, leading to slowed or obstructed microvascular flow – called vaso-occlusion. Vaso-occlusion in turn promotes ischemia-reperfusion (I-R) injury (clockwise). (C) HbS polymer formation also leads to red cell membrane damage and hemolysis (counterclockwise), releasing cell-free hemoglobin (Hb) into circulation. Oxygenated Hb (Fe²⁺) contributes to endothelial dysfunction by consuming nitric oxide (NO) and producing nitrate (NO₃⁻) and methemoglobin (Fe³⁺). Hb can also undergo Fenton chemistry with H₂O₂, generating hydroxyl radicals (•OH) and additional methemoglobin. Methemoglobin (Fe³⁺) can degrade and release cell-free heme (counterclockwise), a potent erythrocyte-derived DAMP. (D) ROS production, TLR4 activation, NET formation, release of tissue- or cell-derived DAMPs, extracellular DNA, and other yet-undefined mediators generated by cell-free heme or I-R injury can induce sterile inflammation by activating the inflammasome in vascular and immune cells, leading to IL-1β release. WikiPathways WP5607: Erythroid lineage cell differentiation transitions from hematopoietic stem cells (HSCs) through successive erythroid progenitors – burst-forming unit–erythroid (BFU-E), colony-forming unit–erythroid (CFU-E) – then proerythroblasts (proEB), basophilic erythroblasts (basoEB), polychromatic erythroblasts (polyEB), and orthochromatic erythroblasts (orthoEB), then and ultimately to reticulocytes, pyrenocytes, and mature red blood cells (RBCs). Stage-specific transcription factors (GATA1, GATA2), surface markers (CD34, CD45, CD71, CD235a), the onset of hemoglobin expression, and other factors are also depicted.
December 21, 2025 at 9:33 AM
wikipathways.fosstodon.org.ap.brid.gy
November 2025 WikiPathways update: 686 edits by 6 contributors and 10 new pathways in the last month. Accessible via #webservices, #rstats, #PathVisio and #Cytoscape.

Supported by #AWSOpen. https://www.wikipathways.org/index.php/Download_Pathways

#bioinformatics #openscience #opensource
New pathway, Caffeine in blood vessels (WP5601). In vascular smooth muscle, caffeine promotes relaxation by inhibiting myosin light-chain kinase (MLCK) through cAMP elevation and activating myosin light-chain phosphatase (MLCP), reducing myosin–actin interaction. As a nonselective adenosine receptor antagonist, caffeine blocks A₁/A₃ receptors (which normally lower cAMP) and A₂A/A₂B receptors (which normally raise cAMP), thereby altering adenylate cyclase activity. In endothelial cells, caffeine increases intracellular Ca²⁺, activating eNOS to produce nitric oxide (NO), which diffuses to smooth muscle and stimulates cGMP signaling, further promoting vasodilation. Inspired by Figure 1 in Kumar and Lipshultz, 2019. New pathway, KCNQ2 and KCNQ3-related epilepsy (WP5599): Processes involved in KCNQ2 and KCNQ3-related epilepsies. New pathways, GABA and glutamate signalling in epileptogenesis (WP5600): This pathway depicts the epileptogenic mechanisms involving GABA and glutamate signaling. It focuses on transporters and channel proteins that modulate action potential dynamics and their interacting partners, highlighting functional alterations that contribute to epilepsy pathogenesis.
November 12, 2025 at 6:21 AM
wikipathways.fosstodon.org.ap.brid.gy
October 2025 WikiPathways update: 129 edits by 10 contributors and 5 new pathways in the last month. Accessible via #webservices, #rstats, #PathVisio and #Cytoscape. Supported by #AWSOpen. https://www.wikipathways.org/index.php/Download_Pathways

#bioinformatics #openscience #opensource
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www.wikipathways.org
October 11, 2025 at 2:44 PM
wikipathways.fosstodon.org.ap.brid.gy
September 2025 WikiPathways update: 261 edits by 6 contributors and 13 new pathways in the last month. Accessible via #webservices, #rstats, #PathVisio and #Cytoscape. Supported by #AWSOpen. https://www.wikipathways.org/index.php/Download_Pathways

#bioinformatics #openscience #opensource
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www.wikipathways.org
September 12, 2025 at 6:59 PM
wikipathways.fosstodon.org.ap.brid.gy
June 2025 WikiPathways release: 338 edits by 15 contributors and 9 new pathways. https://data.wikipathways.org/

Accessible via #webservices, #rstats, Pathvisio, and Cytoscape.
Depiction of the pathway "Lupus pathogenesis" (WP5559): "Systemic lupus erythematosus (SLE) is an autoimmune disease in which the body’s immune system mistakenly attacks its own tissues. It can lead to lupus nephritis, where the kidneys become inflamed. This causes impaired kidney function and, in severe cases, kidney failure. Lupus has an incidence of around 5 per 100,000 person-years, and affects women much more frequently than men. ..." Depiction of the pathway "Atopic dermatitis mechanism and therapies" (WP5538): "Atopic dermatitis (AD), also known as atopic eczema, arises from a complex interplay of genetic susceptibility and environmental triggers that disrupt the skin barrier. This dysfunction is exacerbated by alterations in the skin microbiome—particularly a reduction in Staphylococcus epidermidis and an overgrowth of Staphylococcus aureus—which increase the skin’s vulnerability to external allergens. Keratinocytes, in response to this disruption, release epithelial-derived cytokines such as thymic stromal lymphopoietin (TSLP), interleukin (IL)-25, and IL-33. These cytokines promote type 2 immune responses by activating group 2 innate lymphoid cells (ILC2s), T helper 2 (Th2) cells, and T follicular helper 2 (Tfh2) cells. ..." Depiction of the pathway "Lupus therapies" (WP5560): "Therapeutic approaches in systemic lupus erythematosus (SLE) increasingly focus on modulating B and T cell activity. B cell–directed therapies involve targeting specific surface receptors such as CD19, CD20, CD22, CD38, and CD40, as well as blocking B cell-related cytokines like BAFF (B-cell activating factor, also known as TNFSF13B). T cell-focused strategies include blocking co-stimulatory pathways (e.g. CD40–CD40L, CD80/86–CD28, ICOS–ICOSL) and inhibiting pro-inflammatory cytokines, including IL-6, IL-12, IL-17, IL-23, and IFN-γ. Inspired by Figure 1 in Su et al. (2024)."
June 11, 2025 at 7:04 PM
wikipathways.fosstodon.org.ap.brid.gy
May 2025 WikiPathways release: 491 edits by 9 contributors and 8 new pathways.

Accessible via #webservices, #rstats, Pathvisio, and Cytoscape.

#biology #bioinformatics
Image of the Visual cycle (WP5534) pathway. https://www.wikipathways.org/pathways/WP5534 Description: "The visual cycle, also known as the retinoid cycle metabolic pathway, regenerates vitamin A derived chromophore 11-cis retinal. This enables visual phototransduction response to light. Inspired by rgd.mcw.edu/rgdweb/pathway/pathwayRecord.html?acc_id=PW:0001005." Image of the Atopic dermatitis mechanism and therapies (WP5538) pathway. https://www.wikipathways.org/pathways/WP5538 Description: "Atopic dermatitis (AD), also known as atopic eczema, arises from a complex interplay of genetic susceptibility and environmental triggers that disrupt the skin barrier. This dysfunction is exacerbated by alterations in the skin microbiome—particularly a reduction in Staphylococcus epidermidis and an overgrowth of Staphylococcus aureus—which increase the skin’s vulnerability to external allergens. Keratinocytes, in response to this disruption, release epithelial-derived cytokines such as thymic stromal lymphopoietin (TSLP), interleukin (IL)-25, and IL-33. These cytokines promote type 2 immune responses by activating group 2 innate lymphoid cells (ILC2s), T helper 2 (Th2) cells, and T follicular helper 2 (Tfh2) cells. These lymphocytes, stimulated by activated Langerhans cells (LCs) and inflammatory dendritic epidermal cells (IDECs), subsequently secrete IL-4, IL-5, and IL-13. IL-4 and IL-13 signal through type I (IL-4Rα / CD132) and type II (IL-4Rα / IL-13Rα1) receptors expressed on B cells, keratinocytes, and sensory neurons, activating the JAK-STAT signaling pathway. Tfh2 cells promote class switching to IgE in B cells, enhancing mast cell and basophil activation via FcεRI. [..]" Image of the Fibrotic response of human trabecular meshwork cells to transforming growth factor-beta 3 (TGFB3 and autotaxin (ENPP2, or ATX) in aqueous humor (WP5545) pathway. https://www.wikipathways.org/pathways/WP5545 Description: "This pathway visualises the potential mechanisms of TGF-β3-mediated ENPP2 (or ATX) expression and fibrotic changes in HTM cells. This pathway was created based on the PFOCR project, https://pfocr.wikipathways.org/figures/PMC9496180__biomolecules-12-01231-g008.html" Image of the Multiple sclerosis mechanism and therapies (WP5540) pathway. https://www.wikipathways.org/pathways/WP5540 Description: "This diagram shows the immunopathogenesis of multiple sclerosis (MS) and therapeutic targets. Available therapies for MS act through a range of mechanisms, including pleiotropic effects, immune cell depletion, inhibition of proliferation, and blockade of cell migration. These intervene at various stages of the inflammatory process. Treatments exert their effects across the peripheral immune system, the blood–brain barrier, and within the central nervous system (CNS) itself. Inspired by Figure 1 in Bierhansl et al. (2022)."
May 11, 2025 at 10:09 AM
wikipathways.fosstodon.org.ap.brid.gy
:mega: April 2025 WikiPathways release: 454 edits by 10 contributors and 9 new pathways.

Accessible via #webservices, #rstats, Pathvisio, and Cytoscape. Supported by AWSOpen https://www.wikipathways.org/download.html

#biology #bioinformatics #openscience
new pathway: "Age-related macular degeneration (WP5526)" "Age-related macular degeneration (AMD) is a leading cause of vision impairment in individuals aged 55 and older in the Western world. This diagram shows metabolic pathway crosstalk in AMD. In AMD, the upregulation of PARP2 expression leads to the downregulation of SIRT1. The depletion of SIRT1, a NAD+-dependent deacetylase, causes acetylation and inactivation of PGC-1α." new pathway: "Retinoid metabolism in retina: healthy vs. RLBP1-deficient (WP5532)" "RLBP1 encodes CRALBP, a cellular retinaldehyde-binding protein that functions as a soluble retinoid carrier in the visual cycle of the eye. Mutations in RLBP1 are linked to recessively inherited conditions, such as Bothnia dystrophy, retinitis pigmentosa, retinitis punctata albescens, fundus albipunctatus, and Newfoundland rod-cone dystrophy." new pathway: "Aspirin prevention of cancer metastasis (WP5518)" "Platelets generate TXA2, which binds to its receptor (TP) on T cells, initiating the activation of ARHGEF1, a guanine exchange factor that promotes the conversion of RhoA from its inactive GDP-bound state to its active GTP-bound form. This RhoA activation inhibits TCR-induced kinase pathways, T cell proliferation, and effector functions, ultimately suppressing anti-metastatic immunity. TXA2 production by platelets is COX1-dependent and can be blocked by aspirin or COX-1 selective inhibitors, which relieve T cells from TXA2-mediated suppression. Inspired by figure 14 in in Yang et al. (2025)."
April 10, 2025 at 6:26 PM
egonw.mastodon.social.ap.brid.gy
very exciting Google Summer of Code project idea around @wikipathways for a great student with a lot of enthusiasm and drive to learn a lot of new things and get things done! https://github.com/nrnb/GoogleSummerOfCode/issues/264

#bioinformatics #openscience
Direct GitHub Integration from PathVisio to WikiPathways · Issue #264 · nrnb/GoogleSummerOfCode
Background WikiPathways (https://www.wikipathways.org/) is a collaborative platform for pathway curation in biology, supporting community-contributed models of biological processes. Recently, WikiP...
github.com
March 20, 2025 at 2:07 PM
wikipathways.fosstodon.org.ap.brid.gy
March 2025 WikiPathways release: 1,297 edits by 6 contributors and 1 new pathway

Accessible via #webservices, #rstats, Pathvisio, and Cytoscape. Supported by AWSOpen https://www.wikipathways.org/download.html

#biology #crohn #bioinformatics #openscience
Human pathway for "IL23 inhibitors in inflammatory bowel disease" (WP5516): "IL23 inhibitor drugs decrease an intestinal immune response that contributes to inflammatory bowel disease (IBD), like Crohn’s disease and ulcerative colitis. IL23 inhibitors like risankizumab (Skyrizi), guselkumab (Tremfya), mirikizumab (Omvoh), and brazikumab (MEDI2070) bind to IL23p19 (a.k.a. p19), the protein product of gene IL23A. That in turn blocks IL23A protein from binding to its receptor in immune cells, preventing the production of downstream inflammatory interleukins, cytokines, and other factors that manifest as IBD. IL-23 plays a crucial role in immune responses, especially in the intestinal mucosa. It has a novel p19 protein subunit, which interacts with the p40 subunit. This interaction activates the STAT4-JAK2 pathway. "
March 10, 2025 at 9:00 PM
wikipathways.fosstodon.org.ap.brid.gy
Februari 2025 WikiPathways update: 313 edits by 10 contributors and 4 new pathways in the last month. Accessible via #webservices, #rstats, Pathvisio, and Cytoscape. Supported by AWSOpen https://www.wikipathways.org/download.html

#biology #bioinformatics #openscience
New pathway "Mitochondrial carrier deficiency associated with shuttle disturbances (WP5507)" https://www.wikipathways.org/pathways/WP5507 "Reflecting the pathway as shown in Figure 43.2 of Blau et al. "Physician’s guide to the diagnosis, treatment, and follow-up of inherited metabolic diseases" New pathway "GIP and GLP-1 function by tissue (WP5509)" https://www.wikipathways.org/pathways/WP5509 "Functions of GIP and GLP-1, two agonists of which are found in the weight-loss drug tirzepatide (a.k.a. Mounjaro). "Pancreatic and exopancreatic function of glucose‐dependent insulinotropic polypepide (GIP) and glucagon‐like peptide (GLP)‐1. GIP acts directly on the endocrine pancreas, bone, fat, gastrointestinal (GI) tract and brain. GLP‐1 acts directly on the endocrine pancreas, gastrointestinal tract, heart and brain." From Figure 2 in https://pmc.ncbi.nlm.nih.gov/articles/PMC4020673/." New pathway: "Vedolizumab therapy for irritable bowel syndrome (WP5512)" https://www.wikipathways.org/pathways/WP5512 "Vedolizumab is a humanized monoclonal antibody with a predominantly gastrointestinal effect. It specifically inhibits leucocyte integrin α4β7, thus preventing its interaction with mucosal vascular addressin cell adhesion molecule 1 (MAdCAM-1), which is involved in the migration of lymphocytes from the blood stream to the intestinal tissue. Vedolizumab is indicated in the treatment of moderate to severe active Crohn's disease and ulcerative colitis in adult patients with poor response, loss of response, or intolerance to conventional treatment or to tumour necrosis factor alpha (TNF-α) antagonists." Inspired by Figure 1 in https://pubmed.ncbi.nlm.nih.gov/31470005/. Vedolizumab is also known by its trade name, Entyvio. New pathway: "NSAIDs mechanism of action (WP5513)" https://www.wikipathways.org/pathways/WP5513 "Nonsteroidal anti-inflammatory drugs (NSAIDs) inhibit the cyclooxygenase isoenzymes COX1 and COX2 with varying degrees of specificity for the COX2 isoform. As a result, the conversion of arachidonic acid to prostaglandins (PGs) is inhibited. The main effects of PGs throughout the body are listed." Inspired by Figure 1 in PMID:32322101.
February 11, 2025 at 6:38 AM
wikipathways.fosstodon.org.ap.brid.gy
January 2025 WikiPathways update: 154 edits by 11 contributors and 5 new pathways in the last month. Accessible via #webservices, #rstats, Pathvisio, and Cytoscape. Supported by AWSOpen https://wikipathways.org/index.php/Download_Pathways

#bioinformatics #openscience #opensource
Redirecting…
www.wikipathways.org
January 28, 2025 at 9:24 AM
egonw.mastodon.social.ap.brid.gy
okay, I set up improving the @wikipathways RDF generation, and end up fixing a regression in @bridgedb and two in PathVisio

#openscience #opensource
Screenshot of my GitHub activity for today, showing 7 commits in 5 repositories, a "wip" pull request or BridgeDb, and two pull requests for PathVisio 3.4.
December 28, 2024 at 4:51 PM
wikipathways.fosstodon.org.ap.brid.gy
December 2024 WikiPathways update: 272 edits by 7 contributors and 11 new pathways in the last month. Accessible via #webservices, #rstats, Pathvisio, and Cytoscape. Supported by AWSOpen. https://wikipathways.org/index.php/Download_Pathways #bioinformatics #openscience #opensource
Redirecting…
www.wikipathways.org
December 21, 2024 at 12:58 PM
egonw.social.edu.nl.ap.brid.gy
okay, that's it for today. Screening shows to red flags on these articles, and tomorrow I will two of them with PathVisio

Help us, help MetaKids, help the kids with metabolic disorders. Donate here: https://www.npo3fm.nl/kominactie/acties/wikipathways-in-actie-voor-metakids

#sr24 #metakids
Steun deze actie! WikiPathways in actie voor MetaKids
Onderzoek naar metabole ziekten begint vaak met het in kaart brengen van de biologische processen die aan deze ziekten ten grondslag liggen. WikiPathways is een belangrijke bron van kennis, waarbij visuele kaarten worden gemaakt van deze biologische processen. Het WikiPathways-team van de Universiteit Maastricht en Maastricht UMC+ is daarom aan de slag gegaan om bestaande kaarten te actualiseren met de meest recente inzichten, ontbrekende kaarten toe te voegen, en de kwaliteit van deze kaarten verder te verbeteren voor onderzoek. **Deelnemers** **Egon Willighagen**: "Tijdens de week van 16-20 december zal ik voor elke gesponsorde euro een minuut besteden aan het verbeteren en uitbreiden van beschrijvingen van biologische processen die betrokken zijn bij metabole ziekten in WikiPathways." **Laura Steinbusch**: "Tijdens de week van 16-20 december zal ik voor elke gesponsorde euro een minuut besteden aan het verzamelen van literatuur welke als basis dienen voor verbeteren en uitbreiden van beschrijvingen van biologische processen die betrokken zijn bij metabole ziekten in WikiPathways. Op vrijdag 20 december fiets ik met het metabole team van het MUMC+ naar het glazen huis om het gedoneerde geld over te dragen aan Serious Request." **Martina Kutmon**: "Tijdens de week van 16-20 december zal ik, tot een maximum van 3 werkdagen, voor elke gesponsorde euro een minuut besteden aan het cureren van pathways en het ontwikkelen van technische verbeteringen die bijdragen aan de nauwkeurigheid en toegankelijkheid van informatie over biologische processen die betrokken zijn bij metabole ziekten in WikiPathways." **Daan van Beek**: "In de week voor de kerst zal ik voor elke gedoneerde euro een minuut (maximaal 3 werkdagen) toewijden aan het bijhouden en controleren van de WikiPathways database alswel het ontwikkelen van les- en infomateriaal over metabole stoornissen en het belang van pathway databases in het onderzoek naar deze stoornissen." **Denise Slenter**: "Tijdens de week van 16-20 december zal ik voor elke gesponsorde euro een minuut besteden aan het creëren van nieuwe computer leesbare modellen van metabole ziektes en een gebruikershandleiding maken voor nieuwe gebruikers van WikiPathways."
www.npo3fm.nl
December 16, 2024 at 9:08 PM
egonw.social.edu.nl.ap.brid.gy
I was just in a chat with two action partners and a third person, with Denise showing them around in PathVisio and pathway drawing.

One question came up: how do we draw in @wikipathways proteins with multiple domains, where the domains can have identifiers, and other entities (other proteins […]
Original post on social.edu.nl
social.edu.nl
December 16, 2024 at 11:27 AM
bridgedb.fosstodon.org.ap.brid.gy
a new BridgeDb metabolite ID mapping database has been released, compatible with PathVisio 3.3: https://doi.org/10.6084/m9.figshare.28030427.v1

To support #sr24 #metakids pathway curation, see https://social.edu.nl/@egonw/113599062833652441
Metabolite BridgeDb ID Mapping Database (20241215)
BridgeDb ID mapping database for metabolites, using HMDB 5.0 (Release of April 2024), ChEBI (Release of December 2024), and Wikidata (15 December 2024) as data sources.
figshare.com
December 15, 2024 at 7:48 PM