Blood calcium imbalance is a silent threat to your heart. Low calcium causes QT-prolongation & life-threatening arrhythmias; high calcium triggers at… #Calcium #Cardiacarrhythmia #Electrolytes #Hypercalcemia #Hypocalcemia

Category: Critical Care Nursing  Hypothermia can induce electrolyte abnormalities and increase the sensitivity to them. Specifically, potassium, magnesium and phosphate depletion could cause serious adverse effects, including life-threatening arrhythmias.

Long-term cocaine use, as well as acute cocaine use, is associated with adverse cardiovascular consequences, including arrhythmias, angina, myocardial infarction, heart failure, and other conditions. ...

Cardiac arrhythmias, characterized by disruptions in the heart's electrical conduction system, can range from benign pauses to critical conditions that increase metabolic risks, including obesity. Types of Heart Rhythm Disorders Clinical practice identifies several primary forms of arrhythmia based on pulse rate and electrical activity: Tachycardia: A resting heart rate exceeding 100 beats per minute, often accompanied by chest pain and weakness. Bradycardia: A heart rate of 50 beats per minute or lower. If paired with fainting or cold sweats, it indicates the heart cannot meet the body's demands. Atrial Fibrillation: Considered the most dangerous form, where chaotic electrical activity can reach 600 impulses per minute, significantly increasing the risk of blood clots and stroke. Extrasystole and Blockages: Extrasystole manifests as premature beats or "skipped" beats, while blockages occur when electrical impulses slow down as they pass through cardiac tissues. Cardiologist Artur Glumskov warns against ignoring symptoms such as sudden vision darkening, shortness of breath, and pressure behind the breastbone. According to Glumskov, every minute without resuscitation reduces the probability of survival by approximately 10%.

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Western University launches the Dr. George J. Klein Shah-Baylis Chair to advance heart rhythm research and patient care.

Abstract figure legend In inherited cardiac arrhythmias and cardiomyopathy, arrhythmias are typically triggered by autonomic nervous system activity. Genetic mutations linked to these conditions caus...

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Cardiac amyloidosis (CA) poses a significant risk for ventricular arrhythmias and sudden cardiac death. Arrhythmic risk differs by subtype: the light-chain variant is often more arrhythmogenic due to

CONCLUSIONS: Thin filament HCM seems to carry a higher risk for ventricular arrhythmias and advanced heart failure compared with thick filament HCM. Integrating gene-level and intragene-level information may improve risk stratification in patients with HCM.

Background Supraventricular arrhythmias are frequent in critically ill patients and landiolol, an ultra-short-acting, highly β1-selective adrenergic blocker, is increasingly used for their management. Its efficacy and dosing have been described, but its early hemodynamic tolerance at the bedside — when intolerance occurs, how it presents, and which patients are at risk — remains poorly characterized, particularly in vasopressor-dependent patients under-represented in existing cohorts. We aimed to describe the hemodynamic tolerance of landiolol and to identify bedside predictors of poor tolerance. Methods We conducted a retrospective single-center study in patients admitted to the intensive care unit who received landiolol for supraventricular arrhythmia between January 2021 and February 2023. Hemodynamic variables, including pulse pressure, were recorded at baseline (H0) and at H1, H2, H4, H6 and H24. A hemodynamic adverse event was defined as a composite of mean arterial pressure below 64 mmHg, bradycardia below 50 bpm, or a more than 50% increase in vasopressor dose, counted only while landiolol was infused. Predictors of poor tolerance were identified by multivariable logistic regression. Results Among 133 patients (median age 68 years; SAPS II 51; sepsis 54.9%; vasopressors 49.6%), a hemodynamic adverse event occurred in 27 (20.3%), mostly within the first six hours. Events consisted of hypotension or vasopressor escalation; no patient developed bradycardia below 50 bpm. A lower diastolic pressure (adjusted OR 1.05 per 1-mmHg decrease, 95% CI 1.01–1.10) and vasopressor use at initiation (adjusted OR 3.00, 95% CI 1.14–7.91) were independently associated with poor tolerance. Heart rate control was achieved without deterioration of arterial pressure, pulse pressure, ventricular function, or tissue perfusion. Conversion to sinus rhythm was more frequent in new-onset than in pre-existing atrial fibrillation (54.4% vs. 27.9%; p = 0.004). Conclusion In critically ill patients, hemodynamic intolerance to landiolol occurred in one-fifth of cases, arose early, and took the form of hypotension or increased vasopressor requirements rather than bradycardia. A low diastolic pressure and vasopressor dependence identified patients at risk, supporting cautious, individualized use with close monitoring during early titration.

Beta-blockers are among the most widely prescribed drugs in the management of both supraventricular and ventricular arrhythmias, and they are frequently regarded as a homogeneous pharmacological class...

Omitting defibrillation testing after S-ICD implantation is safe and noninferior to routine testing for spontaneous ventricular arrhythmias.