In Blossoms In Adversity, Zhang Jing Yi’s family has fallen from grace, and her efforts to reverse the family have scared off all eligible bachelors – except the mysterious Hu Yi Tian!

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This article inter-references Asian North American theorizing of techno-orientalism and queer Asia as method. Specifically, it develops queer techno-orientalism...

當製造業離開中國之後，中國依舊是中國。

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On January 22nd, in Liujia Village, Zhucheng City, Weifang, Shandong Province, State Grid Shandong Electric Power Company successfully completed the first whole-village distributed photovoltaic network safety capability improvement and transformation in China, and passed the on-site test verification of experts from Shandong Electric Power Science and Technology Academy, providing a replicable and popularizable practical example for solving the network safety problems of distributed photovoltaics.

by Yizhun Zeng, Xiaopeng Song, Qi Chen, Yue Gu, Jie Zhang, Tao Zhou, Zhihao Li, Tao Wang, Le Chang, Hongwei Yao, Yan Wang, Liyan Miao, Liujia Qian, Tiannan Guo, Yong Zhang, Sonia Rodriguez-Fernandez, Antonio Vidal-Puig, Ying Xu The suprachiasmatic nucleus (SCN), the central circadian pacemaker, orchestrates daily metabolic rhythms, yet its role in substrate selection and thermogenic adaptation under stress remains insufficiently understood. Here, we show that SCN lesioning abolishes the adaptive suppression of brown adipose tissue (BAT) thermogenesis typically observed during time-restricted feeding in subthermoneutral environments (TRF-STE), a paradigm that imposes concurrent nutrient and thermal stress. Contrary to wild-type responses, SCN-lesioned mice maintain elevated BAT thermogenic activity, despite impaired lipolysis, instead shifting toward glucose-driven heat production. This phenotype is accompanied by sustained sympathetic tone and β3-adrenergic receptor (ADRB3) signaling in BAT. Mechanistically, we identify a SCN-regulated ADRB3-S100B signaling axis underlying this metabolic reprogramming. S100B, a nutrient-sensitive calcium-binding protein, is upregulated in BAT following SCN disruption, where it promotes thermogenesis by stimulating brown adipocyte proliferation and suppressing senescence. Functional studies reveal that S100B is both necessary and sufficient for sustaining BAT thermogenesis under TRF-STE. Furthermore, diverse SCN disruption models, including light-induced circadian arrhythmia, N-Methyl-D-aspartic acid (NMDA) excitotoxicity, and Caspase-3-mediated ablation, consistently elevate S100B expression in BAT, reinforcing its role as a convergent effector of SCN-regulated metabolic adaptation. Thus, in intact animal, the SCN restrains the ADRB3-S100B module, gating BAT thermogenic output in accordance with energetic availability. Disruption of SCN output lifts this restraint, unmasking a latent ADRB3-S100B program that preserves thermogenesis when lipid fuel is limited. These findings reveal a previously unrecognized role of the SCN in governing thermogenic flexibility and fuel partitioning, and position the ADRB3-S100B axis as a potential target for mitigating circadian misalignment and metabolic disease.

这期加入我们的是夏楠和流嘉，ta们是性别研究背景的研究者，也是跨国女权和酷儿实践的参与者。我们聊到：离散华人社群的女权和酷儿运动实践，新自由主义经济和国际秩序对卢旺达性别政治的影响，中国工厂里的酷儿工人日常生活中的公共空间与社会再生产，右翼政治对反殖话语的挪用，酷儿马克思主义，田野中的失败和对失败的接受，以及对未来，改变自己和身边的希望。

这期加入我们的是夏楠和流嘉，ta们是性别研究背景的研究者，也是跨国女权和酷儿实践的参与者。我们聊到：离散华人社群的女权和酷儿运动实践，新自由主义经济和国际秩序对卢旺达性别政治的影响，中国工厂里的酷儿工人日常生活中的公共空间与社会再生产，右翼政治对反殖话语的挪用，酷儿马克思主义，田野中的失败和对失败的接受，以及对未来，改变自己和身边的希望。