Publication date: Available online 3 November 2026 Source: Animal Behaviour Author(s): Bert Foquet, Mario A. Poot-Pech, Alyssa Canova, Vivian A. Peralta Santana, Audélia M.C. Mechti, Adrian A. Castellanos, Gregory A. Sword, Hojun Song

According to findings from a multiyear radiotelemetry study, bigger actually means a better chance of survival when it comes to Iowa’s stocked muskies. 

PDF | Jaú, Zungaro jahu (Ihering, 1898), a large migratory catfish endemic to the rio da Prata basin, has a fragile conservation status and its ecology... | Find, read and cite all the research you ne...

We're studying how we can track the behavior of these skulky, secretive birds. With luck and persistence, we may be able to piece together how the reproductive burden affects females season-to-season ...

Radiotelemetry was used to study regional blood flow distribution in Alaska sled dogs during cross-country runs. Doppler ultrasonic flowmeter transducers were chronically implanted on the coronary, re...

We used drone-based radiotelemetry and multispectral imagery to estimate detection and survival probabilities of blue-winged teal broods in Saskatchewan, Canada. Weekly brood survival probabilities, ...

HCTF believes in nurturing and growing conservation leaders of the future. The HCTF Scholarship Grant Program supports students pursuing a higher education leading to a career in wildlife or fish…

I've attempted to build a Survey123 form that calculates an estimated transmitter location using triangulation based on azimuths and GPS points from 3–5 observer positions. Each observer records: A geopoint (their current location) An azimuth (direction of strongest signal via radio telemetry) Th...

Within the vasculature, pannexin 1 (PANX1) channels in smooth muscle cells (SMCs) regulate -adrenergic constriction and blood pressure. PANX1 channel activity is regulated by phosphorylation at Y198, S205 and Y308 residues, but the physiological significance of these modifications is unknown. Here, we utilize newly developed PANX1 Y198F, S205A and Y308F phospho-dead mutant mice to test physiological changes related to hemodynamics. Radiotelemetry-measured blood pressure was decreased in Y198F, increased in Y308F, but unchanged in S205A mice at baseline. Clonidine-sensitive sympathetic-driven hypertension was observed in all mouse lines except Y198F. Pressure myography of third-order mesenteric arteries revealed -adrenergic contractile responses were decreased in Y198F, slightly enhanced in Y308F, but unchanged in S205A, with responses in Y198F vessels mimicking controls treated with PANX1 inhibitors. To understand signaling changes driving these phenotypes, we performed mesenteric artery bulk RNA sequencing, but found a minimal number of differentially expressed genes between phospho-dead mutants and controls. Similarly, co-immunoprecipitation-mass spectrometry of wildtype or phospho-dead mutant-expressing vascular SMCs revealed few interacting proteins distinct to each PANX1 variant. However, PANX1 channel activity assessments in HEK293T cells expressing the 1D-adrenergic receptor as well as each phospho-dead mutant PANX1 showed that phenylephrine-induced ATP release from Y198F channels was significantly decreased compared to wildtype, but current was unaffected. Conversely, basal and phenylephrine-induced S205A and Y308F currents were reduced, but ATP release resembled controls. Taken together, these findings indicate that distinct PANX1 phosphorylation determines PANX1 metabolite release versus current conducting properties and in turn, regulates physiological outcomes in the vasculature.